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DYSREGULATION OF THE (IMMUNO)PROTEASOME PATHWAY IN MCD
teasome in epilepsy-associated pathologies may have great importance in view of the development of new therapeutic strategies.
Acknowledgements
We acknowledge the Bloemenhove clinic (Heemstede, The Netherlands). We are grate- ful to A.M. Iyer and K.W. Geijtenbeek for technical assistance.
This work was supported by European Union’s Seventh Framework Programme (FP7/2007-2013) under the grant agreement no. 602391 (project acronym EPISTOP; JvS, MF, TS, EA) and grant agreement no. 602102 (project acronym EPITARGET; EvV, EA); the Austrian Science Fund (FWF): project no. J3499 (AM) and TSC research award 2015 form the German Tuberous Sclerosis Foundation (TS).
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