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Supplementary Figure 4 In situ hybridization of β1i and β5i, proteasome subunit immunore- activity in control, focal cortical dysplasia (FCD) type IIb and Tuberous Sclerosis Complex (TSC). Panels A-D: control cortex (A-C) and with matter (B-D); β1i (A-B) and β5i (C-D) Panels E-F (FCD IIb) and panels C-G (TSC) shows strong expression within the dysplastic region with several pos- itive dysmorphic neurons [arrows and inserts in E(a) and F], giant cells [inserts in G and H(a)] and glial cells [inserts (b) in E and H) Scale bar in H: A-H: 80 μm.