Page 15 - Recognizing axial spondyloarthritis - Janneke de Winter
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REFERENCES
1. Costantino F, Talpin A, Said-Nahal R, Goldberg M, Henny J, Chiocchia G, et al. Prevalence of spondyloarthritis in reference to HLA-B27 in the French population: Results of the GAZEL cohort. Ann Rheum Dis 2015;74:689–693.
2. Reveille JD, Witter JP, Weisman MH. Prevalence of axial spondylarthritis in the United States: estimates from a cross-sectional survey. Arthritis Care Res (Hoboken) 2012;64:905–10.
3. Said-Nahal R, Miceli-Richard C, Berthelot JM, Duché A, Dernis- Labous E, Blévec G Le, et al. The familial form of spondylarthropathy: A clinical study of 115 multiplex families. Arthritis Rheum 2000;43:1356–1365.
4. Said-Nahal R, Miceli-Richard C, D’Agostino MA, Dernis- Labous E, Berthelot JM, Duche A, et al. Phenotypic diversity is not determined by independent genetic factors in familial spondylarthropathy. Arthritis Rheum 2001;45:478–484.
5. Rahman P, Inman RD, Gladman DD, Reeve JP, Peddle L, Maksymowych WP. Association of interleukin-23 receptor variants with ankylosing spondylitis. Arthritis Rheum 2008;58:1020–5.
6. Burton PR, Clayton DG, Cardon LR, Craddock N, Deloukas P, Duncanson A, et al. Association scan of 14,500 nonsynonymous SNPs in four diseases identifies autoimmunity variants. Nat Genet 2007;39:1329– 37.
7.
8.
9.
10.
11.
12.
Anderson CA, Boucher G, Lees CW, Franke A, D’Amato M, Taylor KD, et al. Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47. Nat Genet 2011;43:246–252.
Dernis E, Said-Nahal R, D’Agostino M, Aegerter P, Dougados M, Breban M. Recurrence of spondylarthropathy among first-degree relatives of patients: a systematic cross- sectional study. Ann Rheum Dis 2009;68:502–7.
Chandran V, Schentag CT, Brockbank JE, Pellett FJ, Shanmugarajah S, Toloza SMA, et al. Familial aggregation of psoriatic arthritis. Ann Rheum Dis 2009;68:664–667. Kruithof E, Baeten D, Rycke L De, Vandooren B, Foell D, Roth J, et al. Synovial histopathology of psoriatic arthritis, both oligo- and polyarticular, resembles spondyloarthropathy more than it does rheumatoid arthritis. Arthritis Res Ther 2005;7:R569-80.
Vandooren B, Noordenbos T, Ambarus C, Krausz S, Cantaert T, Yeremenko N, et al. Absence of a classically activated macrophage cytokine signature in peripheral spondylarthritis, including psoriatic arthritis. Arthritis Rheum 2009;60:966–75.
Noordenbos T, Yeremenko N, Gofita I, Sande M van de, Tak PP, Caňete JD, et al. Interleukin-17-positive mast cells contribute to synovial inflammation in spondylarthritis. Arthritis Rheum 2012;64:99–109.
GENERAL INTRODUCTION AND OUTLINE
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