Page 33 - Helicobacter pylori and Gastric Cancer: From Tumor microenvironment to Immunotherapy
P. 33

 86. Petersen, C.P., et al., Macrophages promote progression of spasmolytic polypeptide-expressing metaplasia after acute loss of parietal cells. Gastroenterology, 2014. 146(7): p. 1727-38 e8.
87. Buzzelli, J.N., et al., IL33 Is a Stomach Alarmin That Initiates a Skewed Th2 Response to Injury and Infection. Cell Mol Gastroenterol Hepatol, 2015. 1(2): p. 203-221 e3.
88. Goldenring, J.R., Gastric intestinal metaplasia and tamoxifen: can we reverse the inevitable? Dig Dis Sci, 2014. 59(6): p. 1078-9.
89. Choi, E., et al., Expression of Activated Ras in Gastric Chief Cells of Mice Leads to the Full Spectrum of Metaplastic Lineage Transitions. Gastroenterology, 2016. 150(4): p. 918-30 e13.
90. Syu, L.J., et al., Invasive mouse gastric adenocarcinomas arising from Lgr5+ stem cells are dependent on crosstalk between the Hedgehog/GLI2 and mTOR pathways. Oncotarget, 2016. 7(9): p. 10255-70.
91. Yun, J.I., et al., Small molecule inhibitors of the hedgehog signaling pathway for the treatment of cancer. Arch Pharm Res, 2012. 35(8): p. 1317- 33.
92. Yoon, C., et al., CD44 expression denotes a subpopulation of gastric cancer cells in which Hedgehog signaling promotes chemotherapy resistance. Clin Cancer Res, 2014. 20(15): p. 3974-88.
                                 Chapter 1
General introduction
 26
31
1























































































   31   32   33   34   35